This article by Lloyd Pye tackles the one remaining conjecture
regarding the emergence of modern humanity.
That conjecture has a cultural tradition behind it and additional
geological support that has been largely ignored.
What it boils down to is that genetic intervention has occurred at
least twice in human history. The first time occurred at least
200,000 years ago and possibly sooner. This led to the emergence of
some form of modern man by 40,000 years ago.
The second event was the direct seeding of Earth with genetically
prepared colonies of agricultural man along with the critical
toolkit. This was done about 9000 BP with several large colonies
afte4r the effects of the Pleistocene Nonconformity had settled down
and the Holocene became well established.
This obviously means real engineering of the human genome way beyond
any natural explanation. That is pretty evident anyway by simple
inspection but also indicative of our next step. Just how does our
genome differ? As it turns out, the differences are both radical and
actually complete improbabilities using any natural protocol.
It goes without saying that every geneticist has the skills to
disprove the general conjecture by reviewing the data and the claims.
Instead their silence remains.
What I would like to see is this work replicated on the San, the
Pygmies and the Bushmen in particular. They are the populations that
may still have missing changes intact. Other populations should also
be tested and cataloged against this understanding.
All other populations have long since been hybridized away.
The take home from this article is that our genome bears ample
evidence of genetic manipulation whose natural provenance is a total
impossibility.
What About Genetic
Differences?
Nothing supports the
Intervention Theory and its Intragalactic Terraformers quite as much
as the fascinating genetic differences between our human DNA, and
chimp and gorilla DNA; and, since recent recovery, Neanderthal DNA;
and, at some point in the future, hominoid DNA.
As we explained a few
pages ago, the second chromosome in humans is a fusion of the 2nd and
3rd chromosomes in higher primates (HP).
The mainstream claims
it was caused by a rare mutation called a Robertsonian translocation,
which can combine chromosomes end-to-end, telomeres-to-telomeres, to
somehow make them function well in a radically new configuration.
To combine two
chromosomes so they can keep working is such an incredibly complex
series of events, if it were not for mainstreamers having a
desperateneed for that event to be considered plausible, they would
laugh it out of existence.
Let’s try to follow
their logic. Since all HP have 48 chromosomes (24 from each parent)
it seems safe to assume any “common ancestor” (CA) of chimps and
humans had 48 chromosomes.
Let’s assume two CAs
have sex, and somehow in that process, the female’s egg has
undergone a Robertsonian translocation mutation and its 2nd and 3rd
chromosomes have fused into one.
When that mutated egg
meets any normal, 24 chromosome sperm, it will not form a fertilized
zygote . . . or if it does, soon after it will expire. Why? Because
to replicate into more cells, each chromosome must line up with its
pair from the other parent before being duplicated and pulled apart
by fibers to opposite ends of the cell.
Then, the one cell
splits into two. This process is called mitosis, and every one of our
trillions of cells are copied in this manner, one after the other
after the other, from the original one cell.
Now, if we consider a
human-chimp cross, the 2nd human chromosome must line up with two
chimp chromosomes. However, as the contents of the cell first
duplicate and then pull apart, the intricate “dance” between them
will soon end.
Why? One copy of the
human 2nd chromosome and one copy each of the chimp 2nd and 3rd must
somehow safely wind up at one end of the cell, while the other three
copies need to be pulled to the other end before the cell can divide.
In that process the
fibers become confused, and the resulting cells try to keep on
replicating, but that chaos continues until the blastula expires.
So, there is no way a
one-chromosome-short zygote will somehow become a viable fetus.
In addition to the
above, now let’s consider the problems found in telomeres and
centromeres.
Telomeres are the
“caps” found at the ends of chromosomes that gradually reduce
after each cell division. Think of them as a long string of “beads”
on a necklace, and after each division of each body’s trillions of
cells, a bead is lost.
When all of the
telomeres have dropped off, the chromosomes stop replicating and the
organism they support will die from advanced “old age.” Nothing
can stop the slow loss of those beads.
Centromeres are
segments of DNA usually located near a chromosome’s middle, and
they are critical to successful cell division, which is the continual
process of life that has to happen correctly, each time, every time,
or things can go very, very haywire within the organism.
Now, with that in
mind, let’s try to imagine what would happen if a pair of
chromosomes fuse in the way the two primate chromosomes fused to
create the “missing” one in humans.
The fusion puts the
two central telomeres (blue) into the middle where the centromeres
should be, and the new chromosome has a pair of (red) centromeres
when it should only have one, and that one should be where the
telomeres are.
This is a serious
problem because telomeres perform a “stopping” function that is
entirely inappropriate in the middle of a chromosome that is supposed
to be fully functional. Uh-oh!
Even worse, the
centromeres are only useful in cell division, so when that occurs
there will be not one, but two places where it is happening, which
will soon lead to a badly tangled mess.
Clearly, mainstreamers
need multiple miracles to make this scenario plausible . . . and
guess what? The exact array of miracles required has been found
within human chromosome #2!
Traces of two HP
telomeres are found in human chromosome #2, between bases
#114,455,823 and #114,455,838. Those 15 are deactivated in some way
that doesn’t stop the chromosome’s normal functioning. They have
been neutered!
With the fused
chromosomes, only the middle two telomeres are “deactivated.” The
one at the top end and the bottom one at the other end are not
altered, so their crucial role in cell division (dropping “beads”)
will continue unhindered. How amazing is that? How . . .
coincidental?
As for leaving two
centromeres where only one can function, guess what? One of those
seems also to have been deactivated, so that normal cell division can
proceed successfully! Wow!
The sequential
precision of this incredible, one-in-trillions fusion forces us to
describe it as yet another of the many miracles the mainstream always
seems to be blessed with. Incredible!
The Big Kahuna of
Human Genetics
While the fusion
“miracles” torture credibility for anyone except mainstreamers,
believe it or not we find several more in other chromosomes in the
human genome! These are inversions.
An inversion can occur
when a segment of any chromosome is sliced into, top and bottom, and
then pulled out, inverted, and put back into its original place, but
with a “flipped” orientation.
According to
textbooks, inversions are caused by “ionizing radiation” that
causes the genetic bonds of chromosome’s to “temporarily” break
loose, during which inversion occurs, followed by a reinsertion.
These are rare, but verifiable.
Also consider that any
two chromosomes might accidentally become “entangled,” and the
result is the brief tearing loose of one segment that then inverts
and moves back into its place.
The beauty of this is
that every inversion is unique, and if passed on creates a landmark
DNA signpost which cannot be reversed back to normal in future
generations that carry it. It also works to disprove Darwin, as we
shall see.
In theory, while an
inversion changes the order of the alleles that comprise chromosomes
and genes, the overall makeup of both will remain unharmed as long as
every gene temporarily segregated from the chromosome is retained in
the process of inversion and reinsertion.
Now, brace yourself
for this: The genome of every human carries nine of those
“miraculous” inversion/insertions that are not found in any of
the corresponding chimp chromosomes! They are located in these human
chromosomes:
1, 4, 5, 9, 12, 15,
16, 17, and 18!
According to Darwinian
evolutionary gospel, this means that at some point after the
proto-humans and chimps split from their supposed common ancestor,
the first of the 9 inversions occurred, eventually to be followed by
8 more.
For example’s sake,
let’s assume that the first occurred in chromosome #1, at 5 mya.
One of the new proto-humans carrying those fused chromosomes gives
birth to a child with an inversion/insertion not carried by chimps in
chromosome #1. At 5 mya. Simple enough.
Now that child must
run the gamut of infant and child mortalities to reach maturity. It
does, and then finds a mate. Unlike the chromosome fusion case, which
won’t allow offspring with a partner having a loose chromosome,
inversions can be passed on with a partner who lacks it.
In each pairing, the
offspring will have a one-in-two chance of inheriting the new
inversion. The same will hold true for their offspring if they carry
it, so their odds of passing it to any one of their children would be
50% - 50%.
Now imagine some
astronomical odds. What the above means is that somehow the
individual with the insertion in chromosome #1 produced a line of
offspring that passed it down to every descendant member of its
species to become a part of nearly 7 billion humans alive today!
Since today we all
have an identical insertion in our #1 chromosome, it means the
insertion had to start at some point with a mutation in one of us who
somehow bequeathed it to the rest of us.
This mutation, whether
it did something good for the individual who had it, or bad, or
nothing at all, would, according to Darwinists, create an aberration
that mushroomed out into humanity like a nuclear bomb. Now hold that
thought.
For as unlikely as all
that is, guess what? The mushroom cloud inversion improbability had
to occur in exactly that way for every one of the eight more times it
occurred! That’s right, it is, in fact, massive improbability to a
power of 9!
In a few million
years, on 9 separate occasions, proto-humans were born with a new and
quite distinct inversion mutation that would then be passed on to
everyhuman alive on Earth today. So the odds of that occurring are
enormously more than long, they are beyond imagining!
With all that said,
here is the kicker, the thing that will lay you low if you’re not
ready for it: Each one had to happen in a sequence! If they all
occurred together, it wouldn’t be evolution.
Let’s get clear on
what the mainstream insists had to happen. Inversion #1 occurred and
the genetic lines of all the other proto-humans had to die out. Only
its progeny would live to pass the inversion along to subsequent
generations.
Next, let’s say that
at 4.5 mya the inversion in chromosome #4 occurred, and, of course,
that one has to occur in one of the progeny in whom the first
Now only its progeny,
carrying the inversions in chromosome #1 and #4, can move forward
into the future. All other proto-human lines at 4.5 mya have to die
out in one way or another.
If the mainstream is
right, and the 9 randomly generated inversions occurred in a
Darwinian sequence of gradually accumulating mutations, it couldn’t
happen any other way. However, is there another way? One that makes
more sense?
Of course there is!
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