Neuroscientists just found a hidden protein switch in your brain that reverses aging and memory loss.

Take NADh as a suplement to supoort neuron health.  That is the takehome.  Not so easy of course.  After all it also assists in solving addiction problems as well.  So much for easy.

Please see a naturalpath up to speed with all this.  Your neurons can be helped.

I am personally dealing with a very rare genetic disorder that in my case is old age onset.  Freidrichs Ataxia no less.  Thus we are working with all this directly.  old Age will get me first, but we know so much because young folks do not see 40.  

So here we are using cutting edge science.

Lanson Burrows Jones Jr.Lanson Burrows Jones Jr. • 2ndPremium • 2ndInnovating Now for Infinite Abundance 🌎 | Plasma | Hydrogen | Nuclear | Quantum Tech | Sustainability | Upcycling | Deep Tech | Coherence Engineering | Datacenter | Utilities HyperScaler PolymathInnovating Now for Infinite Abundance 🌎 | Plasma | Hydrogen | Nuclear | Quantum Tech | Sustainability | Upcycling | Deep Tech | Coherence Engineering | Datacenter | Utilities HyperScaler Polymath2d • Edited •

2 days ago • Edited • Visible to anyone on or off LinkedInFollow

Neuroscientists just found a hidden protein switch in your brain that reverses aging and memory loss. A new Nature Aging study just shattered the story of brain aging being inevitable. Researchers at UCSF discovered a single protein—FTL1 (ferritin light chain 1)—that acts like a on/off switch inside the hippocampus, the seat of learning and memory.

When scientists blocked FTL1 in aged mice, something extraordinary happened:

• Neurons sprouted new branches

• Synapses thickened

• Memories sharpened

The old brains didn’t just stop aging.

They became younger.

The reverse was also true: increase FTL1 in young mice, and their neurons collapsed into early aging. Dendrites shrank. Cognition declined. Their brains aged on fast-forward.

This is not about killing neurons.

It’s about the electrical and metabolic complexity that makes memory possible: dendritic architecture, iron regulation, and hippocampal energy.

And when the protein switch was flipped, decline wasn’t slowed.

It was reversed.

“It is truly a reversal of impairments … much more than merely delaying or preventing symptoms.” — Saul Villeda, UCSF

The data showed:

• FTL1 rises with age, correlating with memory loss

• Reducing FTL1 restored dendritic branching and synaptic signaling

• Overexpressing FTL1 triggered oxidative stress and synaptic collapse

• Boosting NADH metabolism rescued cognition, even in high-FTL1 states

This discovery reframes aging itself.

It isn’t just wear and tear.

It is programmable biology.

A hidden code.

A protein switch waiting to be turned.

The UCSF study used NADH in a targeted way inside neurons to rescue FTL1-induced decline. This Supplement has shown to support general brain energy metabolism, but they are not the same as directly switching off FTL1 like this protein in study but are avaliable today! NADH (nicotinamide adenine dinucleotide + hydrogen) is already available as a supplement. It’s essentially the reduced form of NAD⁺, the critical molecule your cells use for energy metabolism, mitochondrial function, and brain activity.

Imagine what this could mean for Alzheimer’s, dementia, Parkinson’s.

For every family who has watched a loved one fade away.

For decades we believed aging was inevitable.

That memory fades because neurons wear out.

That decline is irreversible.

If memory loss is not destiny but design—

then design can be rewritten.

Neuroscience has just handed us a blueprint.

The only question now is: what doors will we dare to open?

Peer Reviewed sources in first comment 👇

Activate to view larger image,