The developing protocol for dealing directly with cancer is to
induce an alkaline blood environment. This can be done directly with
baking sodas, and better yet with magnesium bicarbonate. Indirectly
we can do this with a vegan diet.
Regardless, at least get the sugar and meat well out of your diet as
this drives the acid environment.
This gets us deep into the known science and helps us build a pretty
clear working model. There will be improvements, but this is today
well ahead of anything we have. In the end though, it still must
work for the dying.
Using this knowledge is inherently safe, but if you decide to apply
this knowledge, please get pH strips from your pharmacy. You need
to know that it is working and to judge just how much bicarbonate you
need to ingest. Too much may not helpful either I am sure but I am
more interested in been able to confirm a continuing background of
alkalinity.
If you use this, please email your experiences so we can share any
knowledge.
Mitochondrial
Medicine
Posted by STAFF -
Matheus on May 23, 2013
Dr. Terry Wahls
learned how to reverse accelerating multiple sclerosis using an
evidence-based, functional medical approach focusing on
orthomolecular nutrients and nutrition. Using the lessons she learned
at the subcellular level, she used diet to cure her MS and get out
of her wheelchair. Her TED talk is quite convincing.
Contemporary medicine
has not understood the basics of mitochondrial physiology. Most
doctors have no idea why magnesium, bicarbonate, selenium and
sulfur are important to the mitochondria. But basic science knows
this so it is a wonder that information does not get out about what
to do to repair and fire up the mitochondrial energy factories of the
cells.
If too many
mitochondria fail, there is nothing that can be done to prevent
death. Any successful treatment can only be to prevent too many
mitochondria from failing.
The mitochondria
are the power stations of our cells. They are as important to our
lives and healths as electrical power stations are to modern
civilization. We just cannot get along without them. If mitochondria
get severely damaged, they die. If cells lose their mitochondria,
they lose their power source, and they die. When enough cells die, we
die.
Mitochondria disease
is not usually life threatening in an emergency sense. There are few
infections that attack the mitochondria though there are poisons like
cyanide which will wipe out our energy stations and kill us without
much fanfare. The mitochondria are extremely sensitive to heavy
metals and general chemical insults. If the mitochondria are denied
the basic nutrition they need to function, they cease to function
normally.
Dr. Majid Ali says,
“Injured mitochondria mutate at much higher rates. Damaged
mitochondria are exhausted mitochondria. Exhausted mitochondria
cannot produce sufficient ATP molecules. An insufficient supply of
ATP molecules means insufficient energy. Insufficient molecular
energy meansclinical chronic fatigue.” These organelles are the
power generators of the cell, converting oxygen and nutrients into
ATP (adenosine triphosphate). ATP is the chemical energy "currency"
of the cell that powers the cell’s metabolic activities. This
process is called aerobic respiration and is the reason animals
breathe oxygen.
Mitochondria work
by generating an electrical potential and a pH gradient across that
inner membrane.
The mitochondrion is different from other organelles[1] because it
has its own DNA and reproduces independently of the cell in which it
is found; an apparent case of endosymbiosis.[2] They really are not
quite us in the sense that their genetic pool is outside of our own
DNA that make us uniquely us. Unlike nuclear DNA, mitochondrial DNA
doesn’t get shuffled every generation, so it is presumed to change
at a slower rate.
Recent
breakthroughs in our understanding of autism and other neurological
diseases are coming through mitochondrial research and it is
important to note that pharmaceutical drugs are mostly mitochondrial
poisons. Mercury is a mitochondrial poison. Data suggest that
moderate levels of mercury administered over an 8 week period can
affect adversely the integrity of mitochondrial membranes.[3] The
field of pediatrics has been perverted by the vaccines and endless
antibiotics they have administered to their patients. Pediatricians
do the unforgiveable when they inject mercury directly into
children’s veins and that is why I wrote and have always given away
The Terror of Pediatric Medicine.
“The
German cancer researcher Dr. Paul Gerhard Seeger[4] demonstrated in
1938 that in most cases cancer starts in the cytoplasm, the
jelly-like outer part of the cell, and especially in the
energy-producing mitochondria. Here food fragments are normally
oxidized in a series of enzymatic steps called the ‘respiratory
chain’. Seeger showed that in cancer cells this
respiratory chain was more or less blocked, especially at the site of
the important enzyme cytochrome oxidase. Without it the
cell can produce energy only anaerobically like a fungal cell. This
is very inefficient and the resulting overproduction of lactic acid
makes the cell and the whole body overly acidic.
Seeger and others
found that cancer cells utilize only between 5 and 50% of the
oxygen of normal cells. The virulence of cancer cells is directly
proportional to their loss of oxygen utilization, and with this to
the degree of blockage of the respiratory chain. In 1957 Seeger
successfully transformed normal cells into cancer cells within a few
days by introducing chemicals that blocked the respiratory chain.
Seeger’s most
important discovery was the certainty that that certain nutrients,
mainly from the vegetable kingdom, could restore cellular respiration
in low-virulence cancer cells and, with this,transform them
back into normal cells. [
what this clearly means is that the real cure for cancer is to
transform them back into normal cells by whatever means – arclein ]
Seeger’s finding
that cancer originates in the cytoplasm and not in the nucleus was
confirmed by other researchers. Between 1975 and 1977 they repeated
an experiment 93 times in which they replaced the nucleus of a
fertilized mouse egg with the nucleus of a cancer cell. In each case
the egg developed into a healthy, cancer-free mouse and even the
offspring remained cancer-free. Similar results were achieved with
frog eggs.
There
is increasing evidence that impairment of mitochondrial function,
oxidative damage, and inflammation are contributing factors in the
pathogenesis of Parkinson’s disease (PD). There is
substantial evidence that there is a deficiency of activity of the
mitochondrial electron transport chain in PD. Impaired mitochondrial
function and activated microglia may both contribute to oxidative
damage in PD.[5]
Multiple
Sclerosis is the most common inflammatory demyelinating disease of
the central nervous system and is the leading cause of non-traumatic
neurological disability in young adults. Researchers believe that
mitochondria play a key role in chronic axonal loss in this disease.
The mitochondria present within the chronically
demyelinated axons will be functioning at full capacity for many
years but eventually, despite antioxidant defenses, free radical
damage will accumulate and mitochondrial function will become
compromised. ATP concentration within the axon will decrease and the
effect on axonal function will be profound.[6]
Tissue
damage in 2,4,6-trinitrobenzene sulfonic acid (TNBS)-induced colitis
is accompanied by the arrest of mitochondrial respiration, loss
of mitochondrial DNA, and the expression of nuclear-encoded
mitochondrial proteins. Selenium effectively protects
colon mitochondria prevented inflammatory and necrotic changes.
Selenium in a high dose is therefore a potential therapeutic agent in
inflammatory bowel disease.[7]
Nearly a century of
scientific research has revealed that mitochondrial dysfunction is
one of the most common and consistent phenotypes of cancer cells. A
number of differences in the mitochondria of normal and cancer cells
include differences in mitochondrial metabolic activity, molecular
composition of mitochondria and mtDNA sequence, as well as in
alteration of nuclear genes encoding mitochondrial
proteins. Mitochondria and Cancer, edited by Keshav K. Singh and
Leslie C. Costello, presents thorough analyses of mitochondrial
dysfunction as one of the hallmarks of cancer.
Dr. Michael R. Eades
says, “As the high-energy electrons are passed along down the inner
mitochondrial membrane they occasionally break free. When they break
free, they become free radicals. These rogue free radicals can then
attack other molecules and damage them. Because these free radicals
are loosed within the mitochondria, the closest molecules for them to
attack are the fats in the mitochondrial membranes. If enough of
these fats are damaged, the membrane ceases to work properly. If
enough of the membrane doesn’t work, the entire mitochodrium is
compromised and ceases functioning. If enough mitochondria bite the
dust, the cell doesn’t work and undergo apoptosis, a kind of
cellular suicide. This chronic damage and loss of cells is the basic
definition of aging.”
Sulfur is one of the
basic elements of life. In fact, sulfur is the fourth most abundant
mineral in the body. The proper acid alkaline balance of the body
cannot be maintained without it.
Sulfur is a main
source of antioxidants in the mitochondria. Sulfur has a vital
relationship with protein, since sulfur is found in the amino acids
methionine, cystine, and cysteine. Thus, these amino acids are known
as the sulfur bearing amino acids which are considered the building
blocks of protein.
Magnesium
Bicarbonate
Magnesium
functions as a bicarbonate co-transporter into cells. And bicarbonate
acts as a transporter of magnesium into the mitochondria. Magnesium
influx is linked with bicarbonate transport according to the Dietary
Reference Intakes guide from the Institute of Medicine. Magnesium
transport into or out of cells requires the presence of
carrier-mediated transport systems. ATPase reaction has a broad
pH optimum centering on neutral pH, with little significant activity
above pH 9.0 or below pH 5.5. Thus anything that moves us
from overall acid conditions toward alkaline that recover the neutral
zone is going to enhance cell metabolism via mitochondrial
optimization.
If cancer is a
mitochondrial disease it can be treated directly with a cocktail of
magnesium bicarbonate,sulfur and selenium and it would probably be a
good idea to supplement strongly with iodine as well. All of
these basic cellular substances will work together to repair damaged
cells and their mitochondria and fire them up for full activity.
In the near future it
will be magnesium bicarbonate available in concentrated form that is
going to turn the world of oncology upside down. It may be a year
before production can make this compound in concentrated form widely
available to the public. Presently it is available to only a precious
few but magnesium bicarbonate water is available in a few bottled
waters like Noah’s in California and Unique Water in Australia.
Within a few weeks I will be introducing a gravity fed Berkey-type of
water filter, which will be the first in the world to produce
magnesium bicarbonate water in peoples’ homes.
[1]
ORGANELLES mean little organs. Basically this means that organelles
have specific roles to play in how cells Gunther, 1003; Romani et
al., 1993work just like organs help the body to function properly as
a whole. Some organelles are separated from the rest of the cell by
lipid bi-layers similar in structure to the cell membrane.
[2]
The endosymbiotic theory concerns the origins of mitochondria and
plastids (e.g. chloroplasts), which are organelles of eukaryotic
cells. According to this theory, these organelles originated as
separate prokaryotic organisms which were taken inside the cell as
endosymbionts. Mitochondria developed from proteobacteria (in
particular, Rickettsiales or close relatives) and chloroplasts from
cyanobacteria.
[3]
The effects of mercury ingestion on hepatic mitochondrial membranes
of chicks. Poult Sci. 1976 Nov;55(6):2280-4.
[4]
The only book available in English is Seeger, P.G. and S. Wolz:
Successful biological control of cancer by combat against the causes.
Neuwieder Verlagsgesellschaft, Neuwied, Germany 1990. The most
important book is Seeger, P.G: Krebs – Problem ohne Ausweg?
(“Cancer – Problem without Solution?”) Verl. f. Medizin
Fischer, Heidelberg, Germany 1974, 2nd ed 1988
[6]
Mitochondrial dysfunction plays a key role in progressive axonal loss
in Multiple Sclerosis. Medical Hypotheses, Volume 64, Issue 4, Pages
669-677 H.Andrews, P.Nichols, D.Bates, D.Turnbull
[7]
High selenium diet protects against tnbs-induced acute inflammation,
mitochondrial dysfunction, and secondary necrosis in rat colon.
TIROSH Oren ; LEVY Eran; REIFEN Ram; Hebrew University of Jerusalem.
ISSN 0899-9007 2007, vol. 23, no11-12, pp. 878-886
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