This is good news and it may be possible to
establish a test for vulnerability.
Essentially asphyxiation kicks in and the victim fails to respond to
solve the threat by often simply moving leading to full suffocation. Establishing vulnerability should allow a
protective protocol but simply making sure that too close bundling is avoided
can help that I suspect.
Most certainly we will no longer see the parents
blamed somehow which certainly occurred in the early years of recognizing this
problem.
It also sounds like a fairly simple developmental
shortcoming that may respond to stimulation easily which is superior to waiting
for maturation to do the job. This is
one flaw you definitely grow out of.
Researchers may have found a cause for
Sudden Infant Death Syndrome
(KTVI) – Sudden Infant
Death Syndrome (SIDS) is responsible for about 2,000 infant deaths in the
United States according to the CDC. It is the leading cause of death
in babies in the first year of life. The Centers for Disease Control
and Prevention defines the SIDS as, “deaths in infants less than 1 year of age
that occur suddenly and unexpectedly, and whose cause of death are not
immediately obvious prior to investigation.”
Now, there may be hope
for solving the mystery over why the deaths occur. Researchers from
Boston Children’s Hospital found chemical abnormalities in the brain stems
of the SIDS babies, regardless of their sleep environment, suggests they had
this abnormality before they died.
This is how the study
was conducted according to a press release from Boston Children’s Hospital:
In the end, they
compared 15 infants with SIDS whose deaths were deemed not to involve asphyxia
(group A), 35 SIDS infants whose deaths were possibly asphyxia-related (group
B) and 9 infants who clearly died from other causes (controls). They excluded
the other infants, who either had insufficient data or had evidence of other
clear risk factors for death, such as exposure to drugs or extremes of
temperature.
Brainstem neurochemical
abnormalities—involving serotonin, serotonin receptors, GABA receptors and
14-3-3 (a protein that regulates serotonin)—were found in both group A and
group B. Neurochemical measures didn’t differ significantly between the two
groups, but each group differed significantly from the controls.
“Even the infants dying
in unsafe sleep environments had an underlying brainstem abnormality that
likely made them vulnerable to sudden death if there was any degree of asphyxia,”
Kinney says. “The abnormality prevents the brainstem from responding to the asphyxial
challenge and waking.”
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